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Activation of mitochondrial energy metabolism protects against cardiac failure

Cardiac failure is the most prevalent cause of death at higher age, and is commonly associated with impaired energy homeostasis in the heart. Mitochondrial metabolism appears critical to sustain cardiac function to counteract aging. In this study, we generated mice transgenically over-expressing the mitochondrial protein frataxin, which promotes mitochondrial energy conversion by controlling iron-sulfur-cluster biogenesis and hereby mitochondrial electron flux.

Activation of mitochondrial energy metabolism protects against cardiac failure

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Jen Farmer

Jen Farmer

Executive Director

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