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FARAFARA Cure FA

Cerebellar pathology in Friedreich's ataxia: Atrophied dentate nuclei with normal iron content

BACKGROUND:

In Friedreich's ataxia (FA) the genetically decreased expression of the mitochondrial protein frataxin leads to disturbance of the mitochondrial iron metabolism. Within the cerebellum the dentate nuclei (DN) are primarily affected. Histopathological studies show atrophy and accumulation of mitochondrial iron in DN.

Read More: Cerebellar pathology in Friedreich's ataxia: Atrophied dentate nuclei with normal iron content


About the Author

Jen Farmer

Jen Farmer

Executive Director

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