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Frataxin deficiency unveils cell-context dependent actions of insulin-like growth factor I on neurons

Friedreich’s ataxia (FRDA) is a neurodegenerative disease caused by deficiency of the mitochondrial iron chaperone frataxin (Fxn). FRDA has no cure, but disease-modifying strategies to increase frataxin are under study. Because insulin-like growth factor I (IGF-I) has therapeutic effects in various types of cerebellar ataxia and exerts protective actions on mitochondrial function, we explored the potential Fxn-stimulating activity of this growth factor on brain cells.

Read More: Frataxin deficiency unveils cell-context dependent actions of insulin-like growth factor I on neurons


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Jen Farmer

Jen Farmer

Executive Director

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