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FARAFARA Cure FA

Impaired myocardial perfusion reserve & fibrosis in Friedreich ataxia: a mitochondrial cardiomyopathy with metabolic syndrome

Cardiomyopathy produces significant mortality in patients with Friedreich ataxia (FA), a genetic disorder that produces intra-mitochondrial iron accumulation. We sought to test the hypothesis that abnormal myocardial perfusion reserve and fibrosis represent early manifestations of cardiomyopathy.

Impaired myocardial perfusion reserve and fibrosis in Friedreich ataxia: a mitochondrial cardiomyopathy with metabolic syndrome


About the Author

Jen Farmer

Jen Farmer

Executive Director

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