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Altered Zinc Transport Disrupts Mitochondrial Protien Processing/Import In Fragile X-Associated Tremor/Ataxia Syndrome

Fragile X-associated tremor/ataxia syndrome (FXTAS) is a late-onset neurodegenerative disorder that affects individuals who are carriers of small CGG premutation expansions in the fragile X mental retardation 1 (FMR1) gene. Mitochondrial dysfunction was observed as an incipient pathological process occurring in individuals who do not display overt features of FXTAS (1). 

Altered Zinc Transport Disrupts Mitochondrial Protien Processing/Import In Fragile X-Associated Tremor/Ataxia Syndrome

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