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Flavin Adenine Dinucleotide Rescues the Phenotype of Frataxin Deficiency

Friedreich ataxia is a neurodegenerative disease caused by the lack of frataxin, a mitochondrial protein. We previously demonstrated that frataxin interacts with complex II subunits of the electronic transport chain (ETC) and putative electronic transfer flavoproteins, suggesting that frataxin could participate in the oxidative phosphorylation.

Flavin Adenine Dinucleotide Rescues the Phenotype of Frataxin Deficiency

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